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Pharmacology: Action: Procardin has an antithrombotic action based on its ability to modify various aspects of platelet function, such as platelet aggregation, adhesion and survival, which have been shown to be factors associated with the initiation of thrombus formation.
Procardin modifies platelet function by enhancing prostacyclin effect through a strong inhibition of phosphodiesterase and mild stimulation of adenyl cyclase formation. It has also been shown to inhibit the formation of thromboxane A2 which is known to cause platelet adhesion and aggregation.
